Here at the ACN, we teach students straight line clinical action. We want the low hanging (nutritional) fruit to be picked.
For easy results with our neuro-mechanical care.
B12 is one such fruit, and the results can be mind-bendingly good.
The protocol we have is relatively straight forward, as per the infographic below.
The full protocol pdf is attached and you can get a card copy if you email inhealthsupplements@yahoo.com.
And you can watch the protocol live here, click it👇:
Passcode: JYXt3e5.
The challenge with B12 is the diagnosis, as the blood test isn’t very reliable.
In the UK, in general if you are below 148 pmol/L (200 ng/L), you are low and need B12.
But above that, and most labs and GP’s will say your “normal” and it is not B12 causing your symptoms, which often look like this:
However, I have read the literature and been using B12 in clinic for 15 years, and it is very clear there is a large gray zone of potential CELLULAR DEFICIENCY – there is no clear cut-off point to define deficiency or adequacy.
Bear in mind that blood is the transport to the cells of your brain, muscles, organs etc.
It does not always reflect the cellular levels, and this if the B12 test is low, it is low, but if it is “normal” it can still be low.
It is the clinical symptoms that are most important.
Thus, we say anything under 500 pmol/L (677 ng/L) total B12 with symptoms we say ALWAYS GIVE A TRIAL OF B12. – because the neurological damage can be permanent.
And hot off the press, comes this incredible research.
They wanted to know:
“Whether B12 levels within the current normal range in a cohort of healthy older adults may be associated with measurable evidence of neurological injury or dysfunction.”
So:
“Methods: We enrolled 231 healthy elderly volunteers (median age 71.2 years old) with a median B12 blood concentration of 414.8 pmol/L. We performed multifocal visual evoked potential testing, processing speed testing, and magnetic resonance imaging to assess neurological status. Moreover, we measured serum biomarkers of neuro-axonal injury, astrocyte involvement, and amyloid pathology.”
The results: Low B12, was associated with visual evoked potential latency delay, processing speed impairment (in an age-dependent manner; standardized, and larger volumes of white matter hyperintensities on MRI.
Remember the MEDIAN B12 blood level was 414 pmol/L, not anywhere close to the NHS 148 pmol/L that would trigger an injection.
That is why we want it over 500 pmol/L and ideally over 650 pmol/L.
How many patients do you see that are over 60, and have less energy they would like?
Have mild loss of cognition? Remember the white lesions they found with lower B12.
This stuff can and does lead to dementia.
Low B12 leads to higher homocysteine and that is directly implicated in dementia.
The good news is our LIPOSOMAL B12 HYDROXO, is as good as an injection (and now back in stock).
I have patients that used to inject themselves and now no longer need to with the liposomal.
I have patients that used to get so tired, with terrible brain fog in the 4-8 weeks before their 3 monthly B12 injection.
And now with LIPOSOMAL B12 and the One a Day Multi Essential, they are good for the 3 months and when they have their jab, nothing happens. They are already at maximum energy.